We haven’t seen any frequency or level on any research so far. Your reference does not answer the issue either: does low frequencies are safer than high frequencies. So far you are only assuming. Will you risk your ears with wishful thinking?
Subsonic sound and some physiological consequences.
(Burdick et al., 1978) indicated
that there may be some permanent threshold shift (PTS) for long term high
level exposure. In one experiment, chinchilla were exposed for three days to
octave band noise at, 100dB, 110dB and 120dB centred on 63Hz. The highest
level led to PTS of up to 40dB at 2kHz in the chinchilla. When human subjects
were exposed to the same low frequency noise at 110dB and 120dB for four
hours, a TTS of about 15dB resulted, extending from low frequencies up to
2kHz. The frequency used by Burdick et al is higher than in the other
experiments and might be expected to have a greater effect. There is an
indication that long-term exposure to very high levels may cause permanent
hearing loss.
The major results were (1) high‐frequency hearing loss to a low‐frequency noise and (2) that noise bands matched within 1 dB
A were not equally hazardous as dictated by damage‐risk criteria. The 63‐Hz noise band produced nearly twice as much PTS as the 1000‐Hz noise band.
Persson and Rylander (1988) surveyed all the 284 local authorities in
Sweden with respect to complaints from heat pumps, heavy traffic and fan and
ventilation installations.
Exposure to 6 and 16 Hz levels at 10 dB
above the auditory threshold have been associated with a reduction in
wakefulness (Landström and Byström, 1984). It has also been possible to
confirm that the reduction on wakefulness is based on hearing perception since
deaf subjects have an absence of weariness (Landström, 1987).
hus although exposure to infrasound
at the levels normally experienced by man does not tend to produce dramatic
health effects, exposure above the hearing perception level will produce
symptoms including weariness, annoyance, and unease. This may precipitate
safety concerns in some environmental and many work situations (Landström
and Pelmear, 1993)
The primary effect of infrasound in humans appears to be annoyance.
(Andresen and Młller, 1984; Broner, 1978a; Młller, 1984)
Beginning at 127 to 133dB, pressure sensation is experienced in the middle ear (Broner 1978a). Regarding potential hearing damage Johnson (Johnson, 1982) concluded that short periods of continuous exposure to infrasound below 150dB are safe and that continuous
exposures up to 24 hours are safe if the levels are below 118dB.
Lidstrom (Lidstrom, 1978) found that long term exposure of active aircraft pilots to infrasound of 14 or 16Hz at 125dB
produced the same changes. Additional findings in the pilots were decreased
alertness, faster decrease in the electrical resistance of the skin compared to
unexposed individuals, and alteration of hearing threshold and time perception.
The ability of infrasound (5 and 16Hz at 95dB for five minutes) to alter body sway responses suggested effects on inner ear function and balance (Tagikawa et al., 1988).
However, Evans (Evans and Tempest, 1972) examining the effect of infrasonic environments on human behaviour found that 30% of normal subjects exposed to tones of 2 – 10Hz through earphones at SPLs of 120 – 150Hz had nystagmus within 60 seconds of exposure to the 120dB signal, with 7Hz being most effective in causing it. Higher intensities
resulted in faster onset of nystagmus, but there were no complaints of
discomfort from any of the subjects at any SPL.
Subsequently, Johnson (Johnson, 1975), who investigated nystagmus in many experiments under different conditions with aural infrasound stimulations from 142 to 155dB had
negative results. For example, an investigator stood on one leg with his eyes
closed, listening aurally to 165dB at 7Hz and 172dB at 1 to 8Hz (frequency
sweep) without effect
Alekseev (Alekseev et al., 1985) exposed rats and guinea pigs (5 test animals,
2 controls per group) to infrasound (4 to 16Hz) at 90 to 145dB for 3 h/day for
45 days; and tissues were collected on days 5, 10, 15, 25, and 45 for
pathomorphological examination. A single exposure to 4 to 10 Hz at 120 to
125dB led to short-term arterial constriction and capillary dilatation in the
myocardium. Prolonged exposure led to nuclear deformation, mitochondrial
damage and other pathologies. Effects were most marked after 10 to 15Hz
exposures at 135 to 145dB. Regenerative changes were observed within 40
days after exposure.
Gordeladze (Gordeladze et al., 1986) exposed rats and guinea pigs (10
animals per group) to 8Hz at 120dB for 3 h/day for 1, 5, 10, 15, 25, or 40 days.
Concentrations of oxidation-reduction enzymes were measured in the
myocardium. Pathological changes in myocardial cells, disturbances of the
microcirculation, and mitochondrial destruction in endothelial cells of the
capillaries increased in severity with increasing length of exposure. Ischemic
foci formed in the myocardium. However, changes were reversible after
exposure ceased.
Male rats (10 /group) exposed to infrasound (8Hz) at 100 and 140dB for 3
h/day for 5, 10, 15, or 25 days showed constriction of all parts of the
conjunctival vascularture within 5 days (Svidovyi and Kuklina, 1985). Swelling
of the cytoplasm and the nuclei of the endotheliocytes accompanied the
decrease in the lumen of the capillaries. The capillaries, pre-capillaries, and
arterioles became crimped. Morphological changes were reported in the
vessels after exposure for 10, 15, and 25 days. After 25 days, increased
permeability of the blood vessels led to swelling of tissues and surrounding
capillaries and to peri-vascular leukocyte infiltration. Significant aggregates of
formed elements of the blood were observed in the large vessels.
Morphological and histochemical changes were studied in the hepatocytes of
rats and guinea pigs exposed to infrasound (2, 4, 8, or 16Hz) at 90, 100, 110,
120, 130 or 140dB for 3 h/day for 5 to 40 days (Nekhoroshev and Glinchikov,
1992a). Hepatocytes showed increased functional activity, but exposures for
25 and 40 days induced irreversible changes. Changes were more pronounced
at 8 and 16Hz than at 2 and 4Hz. Exposures impaired cell organoids and
nuclear chromatin. Single exposures did not induce any changes in the
hepatocytes and small blood vessel
(Shvaiko et al., 1984) found that rats exposed to 8Hz at 90, 115, or 135dB
exhibited statistically significant changes in copper, molybdenum, iron, and/or
manganese concentrations in liver, spleen, brain, skeletal muscle, and/or
femur compared to concentrations in the tissues of controls. Practically all
tissues showed significant changes in all the elements for exposures at 135dB.
Changes included elevations and depressions in concentrations. The trends
were consistent with increasing sound pressure except for some tissue copper
values.
Harding GW, Bohne BA.
The relation between total noise-exposure energy, recovery time, or rest during the exposure and amount of hair-cell loss was examined in 416 chinchillas. The exposures were octave bands of noise (OBN) with a center frequency of either 4 kHz at 47-108 dB sound pressure level (SPL) for 0.5 h to 36 d, or 0.5 kHz at 65-128 dB SPL for 3.5 h to 432 d. Recovery times varied from 0 to 365 d … (4) except for the highest exposure levels, the majority of outer hair cell loss from the 4 kHz OBN occurred after the exposure had terminated, while that from the 0.5 kHz OBN occurred during the exposure; and (5) a majority of the inner hair cell loss from both OBNs occurred post-exposure.
(Nishimura et al., 1987) suggested from experiments on animals that
infrasound influences the rat’s pituitary adreno-cortical system as a stressor,
and that the effects begin at sound pressure levels between 100 and 120 dB at
16Hz. The concentration of hormones shows a slight increase with exposure to
infrasound. In the task performance a reduction was seen in the rate of
working. It seems probable that concentration was impaired by infrasound
exposure.
(Nekhoroshev and Glinchikov, 1992b) exposed rats and guinea pigs (3 per sex
per dose level) to 8Hz at 120 and 140dB for 3 hours or 3 h/day for 5, 10, 15,
25, or 40 days and they showed changes in the heart, neurons, and the
auditory cortex increasing in severity with increasing length of exposure. The
presence of hemorrhagic changes are attributed mostly to the mechanical
action rather than to the acoustic action of infrasound. They suggested that the
changes in the brain may be more important than in the ears
Histopathological and histomorphological changes were determined in the
lungs of male albino mice exposed to infrasound (2, 4, 8, or 16Hz) at 90 to
120dB for 3 h/day for up to 40 days (Svidovyi and Glinchikov, 1987). After
prolonged exposure to 8 Hz at 120 dB sectioned lungs revealed filling of acini
with erythrocytes and thickening of inter-alveolar septa; after prolonged
exposure to 8 and 16Hz at 140dB sectioned lungs revealed ruptured blood
vessel walls, partially destroyed acini, and induced hypertrophy of type-II cells.